est that the observed differences in consumption could be caused by another gene or group of genes that are located near the COMT gene. The interactions of theses genes could cause higher alcohol consumption, and not simply low activity COMT genes.The subjects of this study were not addicted to alcohol, and so, by definition, when and how much they decided to drink was a matter of personal choice. Therefore, if the study is to suggest that the COMT gene influences the choices and conscious thoughts that people have, we must be extremely cautious. A conscious decision, as the social drinker makes, is a very complex process that is difficult to attribute to genetics.The fourth study which was the one that dealt with the alcohol dehydrogenase genes, deals with the findings of previous studies that the ADH3 gene and its varieties are associated with a deterrence to alcohol. This is because they are observed in higher frequencies among controls than alcoholics. The findings of this study are similar to those of previous studies in terms of data, but the interpretation is different due to differences in the statistical work, as well as more informed knowledge about the actual chromosomal location of the involved genes. The study suggests that all the ADH genes are located close to each other on one arm of chromosome 4, and so they are inherited in suite rather than individually. This shows that many studies oversimplify genetic causation. Previous studies had returned certain results because they failed to understand the way in which the genes they studied were inherited, and how they interact with other genes. This shows how lack of complete information and differences in statistical work can change the results of the study.The next study differs in that it deals with rats instead of humans, but there is a very important point that can be made through its findings. The study administered alcohol and cineole to certain rats through their moth...
