ntaneously release retroviruses.(20) The appearance and yield can be increased by (i) mitogenic stimulation;(58) (ii) co-cultivation techniques;(59) (iii) cultivation of cells with supernatant from non-virus producing cultures.(60) According to one eminent retrovirologist, George Todaro, "the failure to isolate endogenous viruses from certain species may reflect thelimitation of in vitro cocultivation techniques";(61) 2. Gallo's team, like everybody else: (i) "isolated HTLV-III (HIV)" from cell cultures; (ii) "isolated HTLV-III" from mitogenically stimulated, activated cell cultures; 3. In addition, Gallo and his colleagues also used co-cultivation techniques; 4. The first "HTLV-III isolation" was from the HT (H4, H9, H17) cell line. Reading Gallo and his colleagues' first paper, one surmises that the HT cell line was established in Gallo's laboratory. The Gallo inquiry revealed that the HT cell line is in fact HUT78, a cell line established in another laboratory from a patient with mature T4-cell leukaemia, a disease which Gallo claims is caused by the exogenous retrovirus, HTLV-I.(3) If so, then all HT cell cultures, and the clones derived from it, "infected with HTLV-III" or non-infected, and the material from these cultures which bands at 1.16 gm/ml, should contain HTLV-I, and thus RT and retroviral particles. Furthermore, because about 25% of AIDS patients have antibodies to HTVL-I,(1) and the immunogenic proteins of HTLV-I and HIV have the same molecular weights, then approximately 25% of the non-infected HT (H4, H9, H17) cultures in addition to RT and particles, should have, in the Western blot, the same bands as those of the "HTLV-III infected" cultures. Thus, these WBs will erroneously appear positive for HTLV-III. Proof that HTLV-III (HIV) is causally linked to AIDS. Gallo claims, a claim accepted by the vast majority of AIDS researchers, that in the May 1984 Science papers he and his colleagues presented "unambiguous evidence...
